Search results for "population stratification"

showing 6 items of 6 documents

Are Polygenic Risk Scores for Major Mental Disorders Associated with General or Specific Psychosis Symptom dimensions?

2019

Background Psychotic symptoms can be conceptualised as dimensions of psychopathology cutting across diagnostic boundaries. Thus, they might be considered enhanced quantitative phenotypes to relate to genetic variants as summarised by Polygenic Risk Scores (PRSs) for Major Mental Disorders (MMDs), including Schizophrenia (SZ), Bipolar Disorder (BP), and Major Depressive Disorder (MDD). The objectives of this study were to: 1) identify the dimensional structure of symptoms at First Episode Psychosis (FEP), testing whether a bi-factor model statistically fits the conceptualization of psychosis as a single common construct (general psychosis factor) while also recognising multidimensionality (p…

PharmacologyPsychosisOdds ratiomedicine.diseasePopulation stratificationschizophrenia poligenic risk scorePsychiatry and Mental healthNeurologySchizophreniamedicineMajor depressive disorderPharmacology (medical)Neurology (clinical)Bipolar disordermedicine.symptomPsychologyManiaSettore MED/25 - PsichiatriaBiological PsychiatryPsychopathologyClinical psychology
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Bayesian model to detect phenotype-specific genes for copy number data

2012

Abstract Background An important question in genetic studies is to determine those genetic variants, in particular CNVs, that are specific to different groups of individuals. This could help in elucidating differences in disease predisposition and response to pharmaceutical treatments. We propose a Bayesian model designed to analyze thousands of copy number variants (CNVs) where only few of them are expected to be associated with a specific phenotype. Results The model is illustrated by analyzing three major human groups belonging to HapMap data. We also show how the model can be used to determine specific CNVs related to response to treatment in patients diagnosed with ovarian cancer. The …

MaleGenotypeGene DosageHapMap ProjectBiologylcsh:Computer applications to medicine. Medical informaticsPopulation stratificationBayesian inferencePolymorphism Single NucleotideBiochemistry03 medical and health sciencesBayes' theorem0302 clinical medicineStructural BiologymedicineHumansComputer SimulationGenetic Predisposition to DiseaseGenetic TestingCopy-number variationInternational HapMap Projectlcsh:QH301-705.5Molecular Biology030304 developmental biologyGenetic testingGenetics0303 health sciencesModels StatisticalModels Geneticmedicine.diagnostic_testMethodology ArticleApplied MathematicsConfoundingBayes Theorem3. Good healthComputer Science ApplicationsPhenotypelcsh:Biology (General)030220 oncology & carcinogenesislcsh:R858-859.7FemaleDNA microarrayAlgorithmsBMC Bioinformatics
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Suggestive evidence for association of D2S2188 marker (2q31.1) with autism in 143 Sicilian (Italian) TRIO families

2005

We have screened 143 Sicilian (Italian) families with one autistic child to verify, by a linkage disequilibrium approach, the involvement of the 2q31.1 region in the cause of the disease in these families. Our study design includes the use of intrafamilial association to prevent a population stratification bias and ethnic homogeneity of the sample. The results of our analysis provided suggestive evidence of the occurrence of transmission disequilibrium between autism and the D2S2188 polymorphism in Sicilian TRIO families, a finding which provides further and independent support to the hypothesis of the existence of a susceptibility gene (or genes) for autism on chromosome 2q.

Genetic MarkersLinkage disequilibriumDisequilibriumEthnic groupautism ds2188 pcrDiseaseBiologyPopulation stratificationSettore BIO/13 - Biologia ApplicataPolymorphism (computer science)GeneticsmedicineHumansFamilyAutistic DisorderSicilyBiological PsychiatryGenetics (clinical)GeneticsPolymorphism GeneticChromosome Mappingmedicine.diseaselanguage.human_languagePsychiatry and Mental healthChromosomes Human Pair 2languageAutismmedicine.symptomSicilianPsychiatric Genetics
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Shared language, diverging genetic histories: high-resolution analysis of Y-chromosome variability in Calabrian and Sicilian Arbereshe.

2015

The relationship between genetic and linguistic diversification in human populations has been often explored to interpret some specific issues in human history. The Albanian-speaking minorities of Sicily and Southern Italy (Arbereshe) constitute an important portion of the ethnolinguistic variability of Italy. Their linguistic isolation from neighboring Italian populations and their documented migration history, make such minorities particularly effective for investigating the interplay between cultural, geographic and historical factors. Nevertheless, the extent of Arbereshe genetic relationships with the Balkan homeland and the Italian recipient populations has been only partially investi…

0301 basic medicineMaleGenetics; Genetics (clinical)PopulationY-chromosome variabilityPopulationSettore BIO/08 - ANTROPOLOGIAPopulation geneticsGenetic admixtureHomelandgenetics (clinical)Diversification (marketing strategy)Population stratificationChromosomesArticle03 medical and health sciencesGeneticModelsGenetic variationHumansgeneticseducationLanguageeducation.field_of_studygenetics (clinical); geneticsChromosomes Human YY chromosomeModels GeneticMediterranean Regionpopulation geneticsGenetic Variationlanguage.human_languageY-ChromosomeChromosomes Human Y; Female; Humans; Italy; Male; Mediterranean Region; Pedigree; Population; Genetic Variation; Language; Models GeneticPedigreeHuman Population Genetics Y chromosome Arbereshe linguistic minorities genetic isolates admixture simulations micro-evolutionary history Southern Italy030104 developmental biologyGeographyItalyEvolutionary biologylanguageArbereshe linguistic minorityArbreshFemaleSicilianSicily and CalabriaYHumanEuropean journal of human genetics : EJHG
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The relationship between health-related quality of life and melancholic depressive symptoms is modified by brain insulin receptor gene network

2021

AbstractTo investigate whether expression-based polygenic risk scores for the insulin receptor gene network (ePRS-IRs) modifiy the association between type of depressive symptoms and health-related quality of life (HRQoL). This cross-sectional study includes 1558 individuals from the Helsinki Birth Cohort Study. Between 2001 and 2004, the Short Form-36 questionnaire was employed to assess mental and physical components of HRQoL and Beck Depression Inventory (BDI) to assess depressive symptoms. Depressive symptoms were categorized into minimal (BDI < 10), non-melancholic and melancholic types of depression. The ePRS-IRs were calculated for the hippocampal (hePRS-IR) and the mesocorticolim…

DISORDERMaleglukoosiaineenvaihduntaelämänlaatuBody Mass Index0302 clinical medicineQuality of lifeSurveys and QuestionnairesMedicineGene Regulatory NetworksDepression (differential diagnoses)METABOLIC SYNDROME2. Zero hunger0303 health sciencesMultidisciplinarydiabetesDepressionQDiabetesRBrainASSOCIATIONriskitekijätMiddle AgedhumanitiesPREVALENCEINVENTORY-IIMedicineFemalegeneettiset tekijätmasennusQuality of lifeRiskmedicine.medical_specialtySciencePopulation stratificationRisk AssessmentArticle03 medical and health sciencesAntigens CDInternal medicineSadnessHumansGenetic Predisposition to DiseaseMETAANALYSISDepressive symptoms030304 developmental biologyAgedHealth related quality of lifePsychiatric Status Rating ScalesDepressive Disorderbusiness.industryterveydentilaInsulin Receptor GeneBeck Depression InventoryReceptor InsulinPSYCHOMETRIC PROPERTIESCross-Sectional StudiesGene Expression Regulation3121 General medicine internal medicine and other clinical medicinebusinessBody mass indexRESISTANCE030217 neurology & neurosurgery
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The case for strategic international alliances to harness nutritional genomics for public and personal health

2005

Nutrigenomics is the study of how constituents of the diet interact with genes, and their products, to alter phenotype and, conversely, how genes and their products metabolise these constituents into nutrients, antinutrients, and bioactive compounds. Results from molecular and genetic epidemiological studies indicate that dietary unbalance can alter gene-nutrient interactions in ways that increase the risk of developing chronic disease. The interplay of human genetic variation and environmental factors will make identifying causative genes and nutrients a formidable, but not intractable, challenge. We provide specific recommendations for how to best meet this challenge and discuss the need …

Knowledge managementNutritional genomicsBiomedical Researchgenetic association030309 nutrition & dieteticsgenotypeInternational CooperationMedicine (miscellaneous)Variation (Genetics)Human genetic variationmedical researchgene–nutrient interactionsVoeding Metabolisme en GenomicaEatingNutrigenomicsenvironmental factorgenetic variabilityGlobal healthNutritional Physiological PhenomenaHealth diaparitiesimmune function2. Zero hunger0303 health sciencesNutrition and Dieteticsstrategic international alliancesarticleGenomicsdiabetes-related traitsdietary fiberHealth equityMetabolism and Genomics3. Good healthNutrigenomicsmessenger-rnaHealthMetabolisme en Genomica/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingNutrition Metabolism and Genomicshealth diaparitiesmedicine.medical_specialtyResearch programhapmap projectpopulation stratificationheredityphenotypeBiologyEnvironmentStrategic international alliancesnutritional health03 medical and health sciencesGene interactionnutrigenomicsSDG 3 - Good Health and Well-beingVoedingmedicineAnimalsHumanscomplex diseaseshuman030304 developmental biologygene identificationVLAGNutritionnonhumanbusiness.industryGenome HumanPublic healthResearchGenetic Variationpopulation geneticsGene-nutrient interactionscultural factorNutrition PhysiologyBiotechnologyDisease Models AnimalHarnessmolecular geneticsbusinessdietary intakepublic health servicecoronary-heart-diseasecarbohydrate ingestionBritish Journal of Nutrition
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